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Direct myocardial cell damage may occur by disruption of mitochondria and induction of intrafibrillar collagen dysplasia (Melchert and Welder 1995; Sullivan et al 1998). Cellular harm results in fibrosis, which could be a potential nidus for ventricular arrhythmias. Patients commonly develop left ventricular hypertrophy, which can be associated to hypertension, one other adverse impact of AAS. The left anterior artery demonstrates a 95% stenosis after the primary septal branch. Scientific and technological services and analysis and design relating thereto; industrial analysis and research companies; design and development of laptop hardware and software.

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